Vaccine Injury Studies

by in Vaccine Safety January 25, 2014

I am slowly working on the links, so hold tight and I’ll get to them as soon as I can! 🙂  Also, if you have a scientific rebuttal for any of the following studies, I’d be happy to post it with the appropriate study (a rebuttal might already be posted in Vaccine Safety Studies or Vaccine Debate).  It is important to view the science from both sides.

Also….

  • Some of these studies listed are dated from years ago, I get it.  To some, they are “outdated.”  I don’t look at it this way… Yes, science is always evolving and new studies outdate the old ones.  My point by posting the older studies is this: no study should be taken as the “gold standard,” always question, always continue researching… and just because one study says the end outcome makes X, Y, Z valid or invalid, doesn’t mean that a study in the future won’t perhaps have a different outcome…  and just because a study might be dated 1976 doesn’t mean the study is outdated.
  • If you take a look at the Merck package insert for the 2014 MMRII, 90% of the studies cited are from the 1960′s, 70′s, and 80′s…

“The important thing is not to stop questioning. Curiosity has its own reason for existing. One cannot help but be in awe when he contemplates the mysteries of eternity, of life, of the marvelous structure of reality. It is enough if one tries merely to comprehend a little of this mystery every day. Never lose a holy curiosity.”  

~Albert Einstein, US (German-born) physicist (1879 – 1955)

Dr. Green Mom- vaccine injury studies

Vaccination as Contamination

  • “Our findings indicate that vaccinal immunity might facilitate an evolutional event through antigenic selection, genetic mutation among virulent virus populations shed from vaccinated flocks, or both.” http://www.ncbi.nlm.nih.gov/pubmed/24689191
  • “We examined live virus vaccines against measles, mumps, and rubella for the presence of pestivirus RNA or of pestiviruses by reverse transcription PCR. Pestivirus RNA was detected in two measles-mumps-rubella combined vaccines and in two monovalent vaccines against mumps and rubella. Nucleotide sequence analysis of the PCR products indicated that a modified live vaccine strain used for immunization of cattle against bovine viral diarrhea is not responsible for the contamination of the vaccines.” (something else inside them is…) http://www.ncbi.nlm.nih.gov/pmc/articles/PMC264050/
  • “The novel human retrovirus xenotropic murine leukemia virus-related virus (XMRV) is arguably the most controversial virus of this moment. After its original discovery in prostate cancer tissue from North American patients, it was subsequently detected in individuals with chronic fatigue syndrome from the same continent. However, most other research groups, mainly from Europe, reported negative results. The positive results could possibly be attributed to contamination with mouse products in a number of cases, as XMRV is nearly identical in nucleotide sequence to endogenous retroviruses in the mouse genome. But the detection of integrated XMRV proviruses in prostate cancer tissue proves it to be a genuine virus that replicates in human cells, leaving the question: how did XMRV enter the human population? We will discuss two possible routes: either via direct virus transmission from mouse to human, as repeatedly seen for, e.g., Hantaviruses, or via the use of mouse-related products by humans, including vaccines. We hypothesize that mouse cells or human cell lines used for vaccine production could have been contaminated with a replicating variant of the XMRV precursors encoded by the mouse genome.” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3109487/
  • “This overview describes the problems and risks associated with viral contaminations in animal cell culture, describes the origins of these contaminations as well as the most important viruses associated with viral contaminations in cell culture.” (cell cultures used in vaccines) “….contaminations are a serious threat for animal cell cultures and may lead to false results in research, development, and virus screening, to viral contaminations in the biologicals derived from the contaminated cultures and finally to an infection of the treated patient.” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3463984/
  • “Current U.S. requirements for testing cell substrates used in production of human biological products (*VACCINES*) for contamination with bovine and porcine viruses are U.S. Department of Agriculture (USDA) 9CFR tests for bovine serum or porcine trypsin. 9CFR requires testing of bovine serum for seven specific viruses in six families (immunofluorescence) and at least 2 additional families non-specifically (cytopathicity and hemadsorption). 9CFR testing of porcine trypsin is for porcine parvovirus. Recent contaminations suggest these tests may not be sufficient. Assay sensitivity was not the issue for these contaminations that were caused by viruses/virus families not represented in the 9CFR screen. A detailed literature search was undertaken to determine which viruses that infect cattle or swine or bovine or porcine cells in culture also have human host range [ability to infect humans or human cells in culture] and to predict their detection by the currently used 9CFR procedures. There are more viruses of potential risk to biological products manufactured using bovine or porcine raw materials than are likely to be detected by 9CFR testing procedures; even within families, not all members would necessarily be detected……..Cell-culture derived vaccines for human use were developed in the 1950’s. Since fetal calf serum and bovine or porcine trypsin were used in cell culture, the 9CFR tests developed for veterinary use to screen for viruses that can infect cattle and swine were implemented by the authorities regulating human vaccines. However, many viruses not of significant concern to the cattle and swine industry are not addressed by the 9CFR testing. Today, over half a century after cell culture-derived vaccines were initially developed, the human biologics industry is still using the methods specified in the 9CFR regulations for testing FBS and porcine trypsin.” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3206158/
  • “Mycoplasmas in frozen bovine serum were effectively inactivated by gamma-irradiation at 25-40 kGy. The larger viruses tested, respiratory enteric orphan (REO) and Cache Valley virus (CVV), were inactivated completely, while the smaller virus, simian virus type 40, was not inactivated. Gamma-irradiation of bovine-sourced serum is therefore useful for mitigating the risk of introduction of mycoplasmas and many of the viral contaminants found in biologics unprocessed bulk (e.g., CVV, REO virus, epizootic hemorrhagic disease virus). This mitigation strategy is not useful for the smaller viruses (e.g., polyomaviruses, parvoviruses, picornaviruses, caliciviruses).” 2010 http://www.ncbi.nlm.nih.gov/pubmed/21502047
  • “All currently licensed yellow fever (YF) vaccines are propagated in chicken embryos. Recent studies of chick cell-derived measles and mumps vaccines show evidence of two types of retrovirus particles, the endogenous avian retrovirus (EAV) and the endogenous avian leukosis virus (ALV-E), which originate from the chicken embryonic fibroblast substrates. In this study, we investigated substrate-derived avian retrovirus contamination in YF vaccines currently produced by three manufacturers (YF-vax [Connaught Laboratories], Stamaril [Aventis], and YF-FIOCRUZ [FIOCRUZ-Bio-Manguinhos]). Testing for reverse transcriptase (RT) activity was not possible because of assay inhibition. However, Western blot analysis of virus pellets with anti-ALV RT antiserum detected three distinct RT proteins in all vaccines, indicating that more than one source is responsible for the RTs present in the vaccines.” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC140796/
  • “Additionally, a baboon endogenous virus strain M7 was detected, likely due to the monkey cell line in which RotaTeq was produced from.”….. “ The sample of RotaTeq vaccine tested positive for rotavirus A and baboon endogenous virus, as previously reported by Victoria and colleagues [17]. The origin of the baboon endogenous virus is assumed to be related to the African green monkey-derived Vero cell line used in its manufacture and cross-hybridization of its endogenous retroviruses to the baboon endogenous retrovirus probes [17]…….Microarray analysis did not detect PCV from the RotaTeq vaccine, which confirmed the previous results from Victoria et al. that LLMDA detected PCV from Rotarix but did not detect PCV from the RotaTeq vaccine [17]. However, PCV2 in RotaTeq vaccine was detected by PCR assays. RotaTeq contained small PCV-1 and PCV-2 genome fragments but did not contain detectable larger portions of PCV genomes [30]. Studies have shown that the amount of PCV in RotaTeq was about 4000 times lower than the PCV in Rotarix, with the PCV in RotaTeq being barely detectable [25, 31, 32]. A case study by Ranucci et al. has reported that the concentration of PCV-2 DNA fragment in clinical consistency lots was in the range of below limit of detection to 6.4 × 103 copies/mL when measured by QPCR, and that PCV1 was below limit of detection (0.1–0.8 × 103 copies/mL) [30]. ” 2014 http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3980782/
  • Investigations of porcine circovirus type 1 (PCV1) in vaccine-related and other cell lines. “Porcine circovirus type 1 (PCV1) is highly prevalent in swine and was recently reported in some rotavirus vaccines.” 011 Oct 26;29(46):8429-37. Epub 2011 Aug 9. http://www.ncbi.nlm.nih.gov/pubmed/21835219?dopt=Abstract
  • ABORTED FETAL CELL LINES- “In some cases the cell lines that are used might be tumorigenic, that is, they form tumors when injected into rodents. Some of these tumor-forming cell lines may contain cancer-causing viruses that are not actively reproducing. Such viruses are hard to detect using standard methods. These latent, or “quiet,” viruses pose a potential threat, since they might become active under vaccine manufacturing conditions.” http://www.fda.gov/…/biologicsresearchareas/ucm127327.htm
  • ‘Xenotropic murine leukemia virus-related virus (XMRV) is a recently discovered human retrovirus that has been found in both chronic fatigue syndrome & prostate cancer patients. There is a potential safety concern regarding XMRV in cell substrates used in vaccines…’ http://www.fda.gov/…/biologicsresearchareas/ucm127327.htm
  • “vaccines are not standard from one batch to the next. 3. Unless the vaccine is properly prepared and refrigerated, its potency and reactivity varies with shelf life. In fact, the whole question of vaccine detoxification has never been systematically investigated. Listed in order of increasing severity, observed adverse reactions include irritability, persistent, unusually high pitched crying, somnolence, seizures, a shock-like “hypotensive, hyporesponsive” state, and an encephalopathy. Since the neurologic picture is not specific for pertussis vaccination, its temporal relationship to the vaccination is the critical variable for determining causation. “ http://www.ncbi.nlm.nih.gov/pubmed/1981251
  • “However, since vaccine preparation involves the use of materials of biological origin, vaccines are subject to contamination by micro-organisms. In fact, vaccine contamination has occurred; a historical example of vaccine contamination, for example, can be found in the early days of development of the smallpox vaccine. The introduction of new techniques of vaccine virus production on cell cultures has lead to safer vaccines, but has not completely removed the risk of virus contamination. There are several examples of vaccine contamination, for example, contamination of human vaccines against poliomyelitis by SV40 virus from the use of monkey primary renal cells. Several veterinary vaccines have been contaminated by pestiviruses from foetal calf serum.These incidents have lead industry to change certain practices and regulatory authorities to develop more stringent and detailed requirements. But the increasing number of target species for vaccines, the diversity of the origin of biological materials and the extremely high number of known and unknown viruses and their constant evolution represent a challenge to vaccine producers and regulatory authorities.” http://www.ncbi.nlm.nih.gov/pubmed/20456974
  • “Although there is no information regarding the duration of acceptable observation period, 1–3 months may not be long enough for the purpose, considering that it takes 2–6 months for adjuvant oils to induce lupus autoantibodies in mice [8,9,34] and that the oil-induced granulomatous inflammation can last for years.”…….”An important factor to consider in vaccine-induced autoimmunity is the fact that vaccines contain a microbial component (or other type of antigens) and adjuvant [75]. Differentiating adverse reactions caused by these two factors is often difficult, or it can even be a result of the combination of both. Nevertheless, the microbial components are generally considered responsible for adverse reactions and minimum attention has been paid to the potential effects of the adjuvant component. Molecular mimicry of a microbial antigen in a vaccine and a host tissue self-antigen is often considered important [61]. Immune complexes also may be formed following vaccination [61,76], deposit in vascular endothelium and induce vasculitis. Induction of cytokines or shifting cytokine balance may also play an important role.” http://www.ncbi.nlm.nih.gov/pubmed/15194169
  • ‘Xenotropic murine leukemia virus-related virus (XMRV) is a recently discovered human retrovirus that has been found in both chronic fatigue syndrome & prostate cancer patients. There is a potential safety concern regarding XMRV in cell substrates used in vaccines…’ http://www.fda.gov/…/biologicsresearchareas/ucm127327.htm
  • “Repeated immunization with antigen causes systemic autoimmunity in mice otherwise not prone to spontaneous autoimmune diseases. Overstimulation of CD4+ T cells led to the development of autoantibody-inducing CD4+ T (aiCD4+ T) cell which had undergone T cell receptor (TCR) revision and was capable of inducing autoantibodies.” “Systemic autoimmunity appears to be the inevitable consequence of over-stimulating the host’s immune ‘system’ by repeated immunization with antigen, to the levels that surpass system’s self-organized criticality.” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2795160/
  • “Clustering of cases of insulin dependent diabetes (IDDM) occurring three years after hemophilus influenza B (HiB) immunization support causal relationship between immunization and IDDM (insulin dependent diabetes).” http://www.ncbi.nlm.nih.gov/pubmed/12911277
  • Immune complexes also may be formed following vaccination, deposit in vascular endothelium and induce vasculitis. Induction of cytokines or shifting cytokine balance may also play an important role.” http://www.ncbi.nlm.nih.gov/pubmed/15194169
  • “We initiated and funded a collaborative study with Tuomilehto on the effect of the Haemophilus influenzae type b vaccine on type 1 diabetes and found that the data support a causal relation (paper submitted for publication). Furthermore, the potential risk of the vaccine exceeds the potential benefit. We compared a group that received four doses of the vaccine, a group that received one dose, and a group that was not vaccinated. The cumulative incidence of diabetes per 100000 in the three groups receiving four, one, and no doses of the vaccine was 261, 237, and 207 at age 7 and 398, 376, and 340 at age 10 respectively.” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1116914/
  • “Successful induction of antiphospholipid syndrome (APS) in two different non-autoimmune prone mouse strains, BALB/c and C57BL/6, was achieved by tetanus toxoid (TTd) hyperimmunization using different adjuvants (glycerol or aluminum hydroxide), and different adjuvant pretreatments (glycerol or Complete Freund’s Adjuvant (CFA)). APS had different manifestations of reproductive pathology in BALB/c and C57BL/6 mice: fetal resorption (as a consequence of extreme T-cell activation obtained in the course of pretreatment), and lowering of fecundity (as a consequence of polyclonal B-cell stimu/lation), respectively. In BALB/c mice fetal resorption coincided with glycerol and CFA pretreatments, while in C57BL/6 mice lowering of fecundity was most obvious in CFA-A pretreated mice immunized with TTd in aluminum hydroxide. Both molecular mimicry and polyclonal B-cell activation occur in APS induction, with molecular mimicry effects being dominant in BALB/c mice, and polyclonal cell activation being dominant in C57BL/6 mice. Confirmation of molecular mimicry effects, which in the condition of T-cell stimulation generated fetal resorptions in the BALB/c strain, was achieved by passive infusion of monoclonal antibody (MoAb) T-26 specific for TTd and anti-β(2)-glycoprotein I obtained after TTd hyperimunization. High polyclonal B-cell activation in C57BL/6 mice prevented fetal resorption but induced fecundity lowering, as was the case in passive administration of MoAb T-26 in this mouse strain. Passive infusion of anti-idiotypic MoAb Y7 into C57BL/6 mice induced fetal resorptions and confirmed the above suggestion on the protective role of polyclonal B-cell stimulation in fetal resorptions.” http://www.ncbi.nlm.nih.gov/pubmed/22235053
  • “Experimental evidence also shows that simultaneous administration of as little as two to three immune adjuvants can overcome genetic resistance to autoimmunity. In some developed countries, by the time children are 4 to 6 years old, they will have received a total of 126 antigenic compounds along with high amounts of aluminum (Al) adjuvants through routine vaccinations. According to the US Food and Drug Administration, safety assessments for vaccines have often not included appropriate toxicity studies because vaccines have not been viewed as inherently toxic. Taken together, these observations raise plausible concerns about the overall safety of current childhood vaccination programs.” Lupus (2012) 21, 223–230 http://www.ncbi.nlm.nih.gov/pubmed/22235057
  • “These findings are consistent with the hypothesis that immunization with the recombinant hepatitis B vaccine is associated with an increased risk of MS, and challenge the idea that the relation between hepatitis B vaccination and risk of MS is well understood.” http://www.neurology.org/content/63/5/838.abstract
  • “Hepatitis B vaccination does not “generally” increase the risk of CNS inflammatory demyelination in childhood. However, the Engerix B vaccine appears to increase this risk, particularly for confirmed multiple sclerosis, in the longer term.” http://www.ncbi.nlm.nih.gov/pubmed/18843097
  • “Acquired autoimmunity syndromes occur after viral vaccinations. Molecular mimicry is involved in these phenomena as is the necessity for the presence of two chemically complimentary antigens and an immunologic adjuvant. The HLA pattern of the host is also an important factor. The example used to explain these phenomena is demyelinating disease that follows hepatitis B vaccination. The somatic antigen of the hepatitis B virus in the vaccine has chemical complimentarity with the Epstein-Barr virus antigen in the vaccine recipient. The Epstein-Barr virus shows molecular mimicry with human myelin. The immunologic adjuvant is either present in the vaccine or muramyl peptides in the individual who is vaccinated. Why more than one type of autoimmune disease occurs is explained by the fact that specific autoimmune T-cells have been shown to develop clones that attack multiple human tissues.” http://www.ncbi.nlm.nih.gov/pubmed/17630224
  • “Herein, we have described a case of vaccine-associated chronic fatigue syndrome and macrophagic myofasciitis in an individual demonstrating aluminium overload. This is the first report linking the latter with either of these two conditions and the possibility is considered that the coincident aluminium overload contributed significantly to the severity of these conditions in this individual. This case has highlighted potential dangers associated with aluminium-containing adjuvants and we have elucidated a possible mechanism whereby vaccination involving aluminium-containing adjuvants could trigger the cascade of immunological events which are associated with autoimmune conditions including chronic fatigue syndrome and macrophagic myofasciitis.” http://www.ncbi.nlm.nih.gov/pubmed/19004564
  • “Although the exact pathogenesis of the development of KFD following immunization remains unknown, this (IMMUNIZATION) should be added to the list of potential triggers or factors associated with the development of KFD” http://www.ncbi.nlm.nih.gov/pubmed/22476507
  • “Vaccination against 2 avian viruses, the Marek disease virus, and the infectious bursal disease virus, were associated with the emergence of more virulent strains (33). An important role of host immunity in selecting for virulence is also suggested by the co-evolution of the myxomatosis virus and rabbits (34). Furthermore, immune pressure was shown to select for more virulent Plasmodium chabaudi parasites in mice (35). Based on mathematical modeling, vaccines designed to reduce pathogen growth rate and/or toxicity may result in the evolution of pathogens with higher levels of virulence.” http://wwwnc.cdc.gov/eid/article/15/8/08-1511_article.htm
  • “Together, our data suggest that the high level of vaccine failure in Nicaraguan is probably not due to antigenic drift of commonly circulating virus strains nor the emergence of new antigenetically distinct virus strains. Furthermore, our data suggest that the widespread use of the RotaTeq vaccine has led to the introduction of vaccine genes into circulating human RotaViruses” Infect Genet Evol. 2012 Aug;12 http://www.ncbi.nlm.nih.gov/pubmed/22487061
  • “Furthermore, while India has been polio-free for a year, there has been a huge increase in non-polio acute flaccid paralysis (NPAFP). In 2011, there were an extra 47,500 new cases of NPAFP. Clinically indistinguishable from polio paralysis but twice as deadly, the incidence of NPAFP was directly proportional to doses of oral polio received. Though this data was collected within the polio surveillance system, it was not investigated.” http://www.ncbi.nlm.nih.gov/pubmed/22591873
  • “We present evidence that in the Netherlands the dramatic increase in pertussis is temporally associated with the emergence of Bordetella pertussis strains carrying a novel allele for the pertussis toxin promoter, which confers increased pertussis toxin (ptx) production. Epidemiologic data suggest that these strains are more virulent in humans.” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2815961/
  • “The biological properties of poxvirus isolates from skin lesions on dairy cows and milkers during recent exanthem episodes in Cantagalo County, Rio de Janeiro State, Brazil, were more like vaccinia virus (VV) than cowpox virus. PCR amplification of the hemagglutinin (HA) gene substantiated the isolate classification as an Old World orthopoxvirus, and alignment of the HA sequences with those of other orthopoxviruses indicated that all the isolates represented a single strain of VV, which we have designated Cantagalo virus (CTGV). HA sequences of the Brazilian smallpox vaccine strain (VV-IOC), used over 20 years ago, and CTGV showed 98.2% identity; phylogeny inference of CTGV, VV-IOC, and 12 VV strains placed VV-IOC and CTGV together in a distinct clade. Viral DNA restriction patterns and protein profiles showed a few differences between VV-IOC and CTGV. Together, the data suggested that CTGV may have derived from VV-IOC by persisting in an indigenous animal(s), accumulating polymorphisms, and now emerging in cattle and milkers as CTGV. CTGV may represent the first case of long-term persistence of vaccinia in the New World.” http://www.ncbi.nlm.nih.gov/pubmed/11080491
  • “Vaccines are not subject to double blind clinical trials despite the evidence of vaccine-drug interactions and perhaps also of vaccine-vaccine interactions.”“Where is the proof that vaccines are safe? The argument has never been that they are completely safe but that the consequences are less than having the disease. Now it is illustrated that the consequences of intensive vaccination schedules pose a greater risk than could ever have been imagined. This leads to the evolution of new viral strains, an unsurprising development when the environment to which it is exposed is being altered by new proteins, structural variants and ALTERED DNA.” http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3364648/
  • “Thus, we conclude that aP (whooping cough) vaccination interferes with the optimal clearance of B. parapertussis and *enhances the *performance of this *pathogen. Our data raise the possibility that *widespread aP vaccination *can *create *hosts *more *susceptible to B. parapertussis infection.” http://www.ncbi.nlm.nih.gov/pubmed/20200027
  • “Although persons often use vaccination and immunization interchangeably in reference to active immunization (VACCINES), the terms are not synonomous because the administration of an immunobiologic CANNOT be automatically equated with the development of adequate immunity.” http://www.cdc.gov/mmwr/PDF/rr/rr4301.pdf 
  • Annual influenza vaccination affects the development of heterosubtypic immunity. 2012- Annual vaccination of healthy children >6 months of age against seasonal influenza has been recommended by public health authorities of some countries. However, currently used seasonal vaccines provide only limited protection against (potentially) pandemic influenza viruses. Furthermore, we recently hypothesized that annual vaccination may hamper the development of cross-reactive immunity against influenza A viruses of novel subtypes, that would otherwise be induced by natural infection. Here we summarize our findings in animal models in which we demonstrated that vaccination against influenza A/H3N2 virus reduced the induction of heterosubtypic immunity against highly pathogenic avian influenza A/H5N1 virus, otherwise induced by a prior infection with influenza A/H3N2 virus. The reduction of heterosubtypic immunity correlated with reduced virus-specific CD8+ T cell responses. An additional study was performed in humans, in which we collected peripheral blood mononuclear cells from annually vaccinated children with cystic fibrosis (CF) and age-matched unvaccinated healthy control children to study the virus-specific T cell response. An age-related increase of the virus-specific CD8+ T cell response was observed in unvaccinated children that was absent in vaccinated children with CF. These findings highlight the importance of the development of vaccines that provide protection against influenza A viruses of all subtypes. (of course the answer is MORE and BETTER vaccines) http://www.ncbi.nlm.nih.gov/pubmed/22643217 
  • “The combined measles, mumps, and rubella (MMR) vaccine has been successfully administered for >20 years. Because of this, protection by maternal antibodies in infants born to vaccinated mothers might be negatively affected…..Conclusions. “Children of mothers vaccinated against measles and, possibly, rubella have lower concentrations of maternal antibodies and lose protection by maternal antibodies at an earlier age than children of mothers in communities that oppose vaccination. This increases the risk of disease transmission in highly vaccinated populations. ” http://www.ncbi.nlm.nih.gov/pubmed/23661802
  • “Varicella vaccination is less effective than the natural immunity that existed in prevaccine communities. Universal varicella vaccination has not proven to be cost-effective as increased HZ (Herpes Zoster {Shingles increased because of vaccine}) morbidity has disproportionately offset cost savings associated with reductions in varicella disease. Universal varicella vaccination has failed to provide long-term protection from VZV disease.” 2013 PMID: 20642419 http://www.ncbi.nlm.nih.gov/pubmed/22659447

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Aseptic Meningitis and the MMR

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Vaccines and Leukemia/Lymphomas

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Vaccines and Chromosome Changes Leading to Mutations

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Vaccines and Autoimmunity

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Vaccinations and Diabetes

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Other Articles Linking Diabetes to Vaccines

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Vaccines and Nervous System Changes

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Vaccinations and Autism

  • Eggers, C, “Autistic Syndrome (Kanner) And Vaccinations against Smallpox”, Klin Paediatr, Mar 1976, 188(2):172-180.
  • Kiln MR, “Autism, inflammatory bowel disease, and MMR vaccine.” Lancet 1998 May 2;351(9112):1358.
  • Selway, “MMR vaccination and autism 1998. Medical practitioners need to give more than reassurance.” BMJ 1998 Jun 13;316(7147):1824.
  • Nicoll A, Elliman D, Ross E, “MMR vaccination and autism 1998,” MJ 1998 Mar 7;316(7133):715-716.
  • Lindley K J, Milla PJ, “Autism, inflammatory bowel disease, and MMR vaccine.”Lancet 1998 Mar 21;351(9106):907-908.
  • Bedford H, et al, “Autism, inflammatory bowel disease, and MMR vaccine.” Lancet 1998 Mar 21;351(9106):907.
  • Vijendra K. Singh, Sheren X. Lin, and Victor C. Yang, “Serological Association of Measles Virus and Human Herpesvirus-6 with Brain Autoantibodies in Autism,” Clinical Immunology and Immunopathology, Oct 1998, Vol. 89, No. 1, p 105-108. [“None of the autistic children in the study had measles in the past, but all had the MMR” stated David Whalgren.

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Vaccines and Demyelination

  • Herroelen, L et al, “Central-Nervous-System Demyelination After Immunization with Recombinant Hepatitis B Vaccine”, Lancet, Nov 9, 1991, 338(8776):1174-1175.
  • Kaplanski G, Retornaz F, Durand J, Soubeyrand J, “Central nervous system demyelination after vaccination against hepatitis B and HLA haplotype.” J Neurol Neurosurg Psychiatry 1995 Jun; 58(6):758-759.
  • Matyszak MK, Perry VH, “Demyelination in the central nervous system following a delayed-type hypersensitivity response to bacillus Calmette-Guerin.” Neuroscience 1995 Feb;64(4):967-977.
  • Tornatore CS, Richert JR, “CNS demyelination associated with diploid cell rabies vaccine.” Lancet 1990 Jun 2;335(8701):1346-1347.
  • Adams, JM et al, “Neuromyelitis Optica: Severe Demyelination Occurring Years After Primary Smallpox Vaccinations”, Rev Roum Neurol, 1973, 10:227-231.
  • In 1988, Dietrich used MRI to show that developmentally delayed children had alterations in their myelin. Coulter described that central nervous system damage can be exhibited as abnormal behavior of the child. In 1935, Thomas Rivers, experimental allergic encephalitis (EAE) can be the result of a viral or bacterial infection of the nervous system. “The fact of the matter is that it is a matter of record that it was known that vaccination produced encephalitis since 1926.” The authors stated, “In regions in which there is no organized vaccination of the population, general paralysis is rare. … It is impossible to deny a connection between vaccinations and the encephalitis (brain damage) which follows it.” Vaccines have been linked to seizures, convulsions and epilepsy.

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Vaccines and Seizures

  • Hirtz DG, Nelson KB, Ellenberg J H, “Seizures following childhood immunizations”, Pediatr 1983 Jan; 102(1):14-18.
  • Cherry JD, Holtzman AE, Shields WD, Buch D, Nielsen, “Pertussis immunization and characteristics related to first seizures in infants and children,”J Pediatr 1993 Jun;122(6):900-903.
  • Coplan J, “Seizures following immunizations,” J Pediatr 1983 Sep;103(3):496.
  • Barkin RM, Jabhour JT, Samuelson J S, “Immunizations, seizures, and subsequent evaluation,” JAMA 1987 Jul 10;258(2):201.
  • Griffin MR, et al, “Risk of seizures after measles-mumps-rubella immunization,” Pediatrics 1991 Nov;88(5):881-885.
  • Griffin MR, et al, “Risk of seizures and encephalopathy after immunization with the diphtheria-tetanus-pertussis vaccine,” JAMA 1990 Mar 23-30;263(12):1641-1645.
  • Cizewska S, Huber Z, Sluzewski W, “[Prophylactic inoculations and seizure activity in the EEG],” Neurol Neurochir Pol 1981 Sep-Dec;15(5-6):553-557. [Article in Polish]
  • Huttenlocher PR, Hapke RJ, “A follow-up study of intractable seizures in childhood.” Ann Neurol 1990 Nov; 28(5):699-705.
  • Blumberg DA, “Severe reactions associated with diphtheria-tetanus-pertussis vaccine: detailed study of children with seizures, hypotonic-hypo-responsive episodes, high fevers, and persistent crying.”Pediatrics 1993 Jun; 91(6):1158-1165. Vaccinations and Convulsions Citations:
  • Prensky AL, et al, “History of convulsions and use of pertussis vaccine,” J Pediatr 1985 Aug; 107(2):244-255.
  • Baraff LJ, “Infants and children with convulsions and hypotonic-hypo-responsive episodes following diphtheria-tetanus-pertussis immunization: follow-up evaluation,” Pediatrics 1988 Jun; 81(6):789-794.
  • Jacobson V, “Relationship of pertussis immunization to the onset of epilepsy, febrile convulsions and central nervous system infections: a retrospective epidemiologic study,” Tokai J Exp Clin Med 1988;13 Suppl: 137-142.
  • Cupic V,et al, “[Role of DTP vaccine in the convulsive syndromes in children],” Lijec Vjesn 1978 Jun; 100(6):345-348. [Article in Serbo-Croatian (Roman)]
  • Pokrovskaia NIa, “[Convulsive syndrome in DPT vaccination (a clinico-experimental study)],” Pediatriia 1983 May;(5):37-39. [Article in Russian] Vaccinations and Epilepsy Citations:
  • Ballerini, Ricci, B, et al, “On Neurological Complications of Vaccination, With Special Reference to Epileptic Syndromes,” Riv Neurol, Jul-Aug 1973, 43:254-258.
  • Wolf SM, Forsythe A, “Epilepsy and mental retardation following febrile seizures in childhood,” Acta Paediatr Scand 1989 Mar;78(2):291-295.

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Vaccines and Brain Swelling

  • Iwasa, S et al, “Swelling of the Brain in Mice Caused by Pertussis … Quantitative Determination and the Responsibility of the Vaccine”, Jpn J Med Sci Biol, 1985 , 38(2):53-65.
  • Mathur R, Kumari S, “Bulging fontanel following triple vaccine.” Indian Pediatr 1981 Jun;18(6):417-418.
  • Barry W, Lenney W, Hatcher G, “Bulging fontanelles in infants without meningitis.” Arch Dis Child 1989 Apr;64(4):635-636.
  • Shendurnikar N, “Bulging fontanel following DPT” Indian Pediatr 1986 Nov;23(11):960.
  • Gross TP, Milstien JB, Kuritsky JN, “Bulging fontanelle after immunization with diphtheria-tetanus-pertussis vaccine and diphtheria-tetanus vaccine.” J Pediatr 1989 Mar;114(3):423-425.
  • Jacob J, Mannino F, “Increased intracranial pressure after diphtheria, tetanus, and pertussis immunization.” Am J Dis Child 1979 Feb;133(2):217-218.
  • Dugmore, WN, “Bilateral Oedema at the Posterior Pole. Hypersensitivity Reaction to Alavac P injection.” Br J Ophthalmol, Dec 1972, 55:848-849.

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Vaccines and Neurological Damage

  • Nedar P R, and Warren, R J, “Reported Neurological Disorders Following Live Measles Vaccine”, 1968, Ped, 41:997-1001.
  • Paradiso, G et al, “Multifocal Demyelinating Neuropathy after Tetanus Vaccine”, Medicina (B Aires), 1990, 50(1):52-54.
  • Landrigan, PJ, Whitte, J, “Neurologic Disorders Following Live Measles-virus Vaccination”, JAMA, Mar 26, 1973, v223(13):1459-1462.
  • Turnbull, H M, “Encephalomyelitis Following Vaccination”, Brit Jour Exper Path, 7:181, 1926.
  • Kulenkampff, M et al, “Neurological Complications of Pertussis Inoculation”, Arch Dis Child, 1974, 49:46.
  • Strom, J, “Further Experience of Reactions, Especially of a Cerebral Nature in Conjunction with Triple Vaccination”, Brit Med Jour, 1967, 4:320-323.
  • Berg, J M, “Neurological Complications of Pertussis Immunization,” Brit Med Jour, July 5,1958; p 24.
  • Bondarev, VN et al, “The Changes of the Nervous System in Children After Vaccination”, Pediatria, Jun 1969; 48:20-24.
  • Badalian, LO, “Vaccinal Lesions of the Nervous System in Children,” Vop Okhr Materin Dets, Dec 1959, 13:54-59
  • Lorentz, IT, et al, “Post-Vaccinal Sensory Polyneuropathy with Myoclonus”, Proc Aust Ass Neurol, 1969, 6:81-86.
  • Trump, R C, White, T R, “Cerebellar Ataxia Presumed Due To Live Attenuated Measles Virus Vaccine,” JAMA, 1967, 199:165-166.
  • Allerdist, H, “Neurological Complications Following Measles Vaccination”, Inter Symp, Brussels, 1978, Development Biol Std, Vol 43, 259-264.
  • Finley, K H, “Pathogenesis of Encephalitis Occurring With Vaccination, Variola and Measles, Arch Neur and Psychologist, 1938; 39:1047-1054.
  • Froissart, M et al, “Acute Meningoencephalitis Immediately after an Influenza Vaccination”, Lille Med, Oct 1978, 23(8):548-551.
  • Pokrovskaia, Nia, et al, “Neurological Complications in Children From Smallpox Vaccination”, Pediatriia, Dec 1978, (12):45-49.
  • Allerdist, H, “Neurological Complications Following Measles Virus Vaccination. Evaluation of the Cases seen Between 1971-1977″, Monatsschr Kinderheilkd, Jan 1979, 127(1): 23-28.
  • Ehrengut, W et al, “On Convulsive Reactions Following Oral vaccination Against Polio”, Klin Paediatr, May 1979, 191(3):261-270.
  • Naumova, R P, et al, “Encephalitis Developing After Vaccination without a Local Skin Reaction”, Vrach Delo, Jul 1979, (7):114-115.
  • Goswamy, BM, “Neurological Complications After Smallpox Vaccination”, J Ass Phys India, Jan 1969, 17:41-43.
  • Schchelkunov, SN et al, “The Role of Viruses in the Induction of Allergic Encephalomyelitis,” Dokl Akad Nauk SSSR, 1990,315(1):252-255. [Vaccines contain viruses, too]
  • Walker AM, “Neurologic events following diphtheria-tetanus-pertussis immunization,” Pediatrics 1988 Mar;81(3):345-349.
  • Shields WD, et al, “Relationship of pertussis immunization to the onset of neurologic disorders: a retrospective epidemiologic study,” J Pediatr 1988 Nov; 113(5):801-805.
  • Wilson J, “Proceedings: Neurological complications of DPT inoculation in infancy,” Arch Dis Child 1973 Oct; 48(10):829-830.
  • Iakunin IuA, “[Nervous system complications in children after preventive vaccinations],” Pediatriia 1968 Nov; 47(11):19-26. [Article in Russian]
  • Greco D, et al, “Case-control study on encephalopathy associated with diphtheria-tetanus immunization in Campania, Italy,” Bull World Health Organ 1985;63(5):919-925.
  • Ehrengut W at Institute of Vaccinology and Virology, Hamburg, Germany states, “Bias in the evaluation of CNS complications following pertussis immunization are the following: 1) Notifications of post-immunization adverse events, 2) Publications by vaccine producers on the frequency of adverse reactions, 3) Comparison of permanent brain damage after DPT and DT immunization, 4) Pro-immunization, 5) Immunization associated viral encephalitis, 6) Accuracy of statistics, 7) Personal. A review of these points indicates an underestimation of CNS complications after pertussis immunization.”
  • Reference: Ehrengut W, “Bias in evaluating CNS complications following pertussis immunization.” Acta Paediatr Jpn, 1991 Aug; 33(4):421-427.
  • Sienkiewicz D.*, Kułak W., Okurowska-Zawada B., Paszko-Patej G. “Neurologic adverse events following vaccination,” Prog Health Sci 2012, Vol 2 , No1.  FULL TEXT

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Vaccinations and Unexplained Diseases

  • Hiner, E E, Frasch, C E, “Spectrum of Disease Due to Haemophilus Influenza Type B Occurring in Vaccinated Children”, J Infect Disorder, 1988 Aug; 158(2): 343-348.
  • Olin P, Romanus, V, Storsaeter, J, “Invasive Bacterial Infections During an Efficiacy Trial of Acellular Pertussis Vaccines — Implications For Future Surveilance In Pertussis Vaccine Programmes”, Tokai J Exp Clin Med, 1988; 13 Suppl: 143-144.
  • Storsaeter, J, et al, “Mortality and Morbidity From Invasive Bacterial Infections During a Clinical Trial of Acellular Pertussis Vaccines in Sweden”, Pediatr Infect Disorder J, 1988 Sept; 7(9):637-645.
  • Vadheim, CM, et al, “Effectiveness and Safety of an Haemophilus Influenzae type b Conjugate Vaccine (PRP-T) in Young Infants. Kaiser-UCLA Vaccine Study Group,” Pediartics, 1993 Aug; 92(2):272-279. [The vaccines caused fevers, irritability, crying, and seizures, but were declared to be “safe and … effective … “.]
  • Stickl, H, “Estimation of Vaccination Damage”, Med Welt, Oct 14, 1972, 23:1495-1497.
  • Waters, VV, et al, “Risk Factors for Measles in a Vaccinated Population”, JAMA, Mar 27, 1991, 265(12): 1527.
  • Stickl, H, “Iatrogenic Immuno-suppression as a Result of Vaccination”, Fortschr Med, Mar 5, 1981, 99(9);289-292. Vaccine Citations Linking the Vaccine to the “prevented” Disease:
  • Nkowane, et al, “Vaccine-Associated Paralytic Poliomyelitis, US 1973 through 1984, JAMA, 1987, Vol 257:1335-1340.
  • Quast, et al, “Vaccine Induced Mumps-like Diseases”, nd, Int Symp on Immun, Development Bio Stand, Vol 43, p269-272.
  • Green, C et al, “A Case of Hepatitis Related to Etretinate Therapy and Hepatitis B Vaccine”, Dermatologica, 1991, 182(2):119-120.
  • Shasby, DM, et al, “Epidemic Measles in Highly Vaccinated Population”, NEJM, Mar 1977, 296(11): 585-589.
  • Tesovic, G et al, “Aseptic Meningitis after Measles, Mumps and Rubella Vaccine”, Lancet, Jun 12, 1993, 341(8859):1541.
  • Johnson, RH, et al, “Nosocomial Vaccinia Infection”, West J Med, Oct 1976, 125(4):266-270.
  • Malengreau, M, “Reappearance of Post-Vaccination Infection of Measles, Rubella, and Mumps. Should Adolescents be re-vaccinated?” Pedaitric, 1992;47(9):597-601 (25 ref)
  • Basa, SN, “Paralytic Poliomyelitis Following Inoculation With Combined DTP Prophylactic. A review of Sixteen cases with Special Reference to Immunization Schedules in Infancy”, J Indian Med Assoc, Feb 1, 1973, 60:97-99.
  • Landrigan, PJ et al, “Measles in Previously Vaccinated Children in Illinois”, Ill Med J, Arp 1974, 141:367-372.
  • NA, “Vaccine-Associated Poliomyelitis”, Med J Aust, Oct 1973, 2:795-796. Vaccine Failures Citations:
  • Hardy, GE, Jr, et al, “The Failure of a School Immunization Campaign to Terminate an Urban Epidemic of Measles,” Amer J Epidem, Mar 1970; 91:286-293.
  • Cherry, JD, et al, “A Clinical and Serologic Study of 103 Children With Measles Vaccine Failure”, J Pediatr, May 1973; 82:801-808.
  • Jilg, W, et al, “Inoculation Failure Following Hepatitis B Vaccination”, Dtsch Med wochenschr, 1990 Oct 12; 115(41):1514-1548.
  • Plotkin, SA, “Failures of Protection by Measles Vaccine,” J Pediatr, May 1973; 82:798-801.
  • Bolotovskii, V, et al, “Measles Incidence Among Children Properly Vaccinated Against This Infection”, ZH Mikrobiol Epidemiol Immunobiol, 1974; 00(5):32-35.
  • Landrigan, PJ, et al, “Measles in Previously Vaccinated Children in Illinois”, Ill Med J, Apr 1974; 141:367-372.
  • Strebel, P et al, “An Outbreak of Whooping Cough in a Highly Vaccinated Urban Community”, J Trop Pediatr, Mar 1991, 37(2): 71-76.
  • Forrest, JM, et al, “Failure of Rubella Vaccination to Prevent Congenital Rubella,”Med J Aust, 1977 Jan 15; 1(3): 77.
  • Jilg, W, “Unsuccessful Vaccination against Hepatitis B”, Dtsch Med Wochenschr, Nov 16, 1990, 115(46):1773.
  • Coles, FB, et al, “An Outbreak of Influenza A (H3N2) in a Well-Immunized Nursing home Population,” J Am ger Sociologist, Jun 1992, 40(6):589-592.
  • Jilg, W, et al, “Inoculation Failure following Hepatitis B Vaccination,” Dtsch Med Wochenschr, Oct 12, 1990, 115(41):1545-1548.
  • Hartmann, G et al, “Unsuccessful Inoculation against Hepatitis B,” Dtsch Med Wochenschr, May 17, 1991, 116(20): 797.
  • Buddle, BM et al, “Contagious Ecthyma Virus-Vaccination Failures”, Am J Vet Research, Feb 1984, 45(2):263-266.
  • Mathias, R G, “Whooping Cough In Spite of Immunization”, Can J Pub Health, 1978 Mar/Apr; 69(2):130-132.
  • Osterholm, MT, et al, “Lack of Efficacy of Haemophilus b Polysacharide Vaccine in Minnesota”, JAMA, 1988 Sept 9; 260(10:1423-1428.
  • Johnson, RH, et al, “Nosocomial Vaccinia Infection”, West J Med, Oct 1976, 125(4):266-270. Vaccines Causing Another Vaccinal Disease:
  • Basa, SN, “Paralytic Poliomyelitis Following Inoculation With Combined DTP Prophylactic. A review of Sixteen cases with Special Reference to Immunization Schedules in Infancy”, J Indian Med Assoc, Feb 1, 1973, 60:97-99.
  • Pathel, JC, et al, “Tetanus Following Vaccination Against Small-pox”, J Pediatr, Jul 1960; 27:251-263.
  • Favez, G, “Tuberculous Superinfection Following a Smallpox Re-Vaccination”, Praxis, July 21, 1960; 49:698-699.
  • Quast, Ute, and Hennessen, “Vaccine-Induced Mumps-like Diseases”, Intern Symp on Immunizations , Development Bio Stand, Vol 43, p 269-272.
  • Forrest, J M, et al, “Clinical Rubella Eleven months after Vaccination,” Lancet, Aug 26, 1972, 2:399-400.
  • Dittman, S, “Atypical Measles after Vaccination”, Beitr Hyg Epidemiol, 19891, 25:1-274 (939 ref)
  • Sen S, et al, “Poliomyelitis in Vaccinated Children”, Indian Pediatr, May 1989, 26(5): 423-429.
  • Arya, SC, “Putative Failure of Recombinant DNA Hepatitis B Vaccines”, Vaccine, Apr 1989, 7(2): 164-165.
  • Lawrence, R et al, “The Risk of Zoster after Varicella Vaccination in Children with Leukemia”, NEJM, Mar 3, 1988, 318(9): 543-548. Vaccination Citations and Death
  • Na, “DPT Vaccination and Sudden Infant Death – Tennessee, US Dept HEW, MMWR Report, Mar 23, 1979, vol 28(11): 132.
  • Arevalo, “Vaccinia Necrosum. Report on a Fatal Case”, Bol Ofoc Sanit Panamer, Aug 1967, 63:106-110.
  • Connolly, J H, Dick, G W, Field, CM, “A Case of Fatal Progressive Vaccinia”, Brit Med Jour, 12 May 1962; 5288:1315-1317.
  • Aragona, F, “Fatal Acute Adrenal Insufficiency Caused by Bilateral Apoplexy of the Adrenal Glands (WFS) following Anti-poliomyelitis Vaccination”, Minerva Medicolegale, Aug 1960; 80:167-173.
  • Moblus, G et al, “Pathological-Anatomical Findings in Cases of Death Following Poliomyelitis and DPT Vaccination”, Dtsch Gesundheitsw, Jul 20, 1972, 27:1382-1386.
  • NA, “Immunizations and Cot Deaths”, Lancet, Sept 25, 1982, np.
  • Goetzeler, A, “Fatal Encephalitis after Poliomyelitis Vaccination”, 22 Jun 1961, Muenchen Med Wschr, 102:1419-1422.
  • Fulginiti, V, “Sudden Infant Death Syndrome, Diphtheria-Tetanus Toxoid-Pertussis Vaccination and Visits to the Doctor: Chance Association or Cause and Effect?”, Pediatr Infect Disorder, Jan-Feb 1983, 2(1): 7-11.
  • Baraff, LJ, et al, “Possible Temporal Association Between Diphtheria-tetanus toxoid-Pertussis Vaccination and Sudden Infant Death Syndrome”, Pediatr Infect Disorder, Jan-Feb 1983, 2(1): 5-6.
  • Reynolds, E, “Fatal Outcome of a Case of Eczema Vaccinatum”, Lancet, 24 Sept 1960, 2:684-686.
  • Apostolov. et al, “Death of an Infant in Hyperthermia After Vaccination”, J Clin Path, Mar 1961, 14:196-197.
  • Bouvier-Colle, MH, “Sex-Specific Differences in Mortality After High-Titre Measles Vaccination”, Rev Epidemiol Sante Publique, 1995; 43(1): 97.
  • Stewart GT, “Deaths of infants after triple vaccine.”, Lancet 1979 Aug 18;2(8138):354-355.
  • Flahault A, “Sudden infant death syndrome and diphtheria/tetanus toxoid/pertussis/poliomyelitis immunisation.”, Lancet 1988 Mar 12;1(8585):582-583.
  • Larbre, F et al, “Fatal Acute Myocarditis After Smallpox Vaccination”, Pediatrie, Apr-May 1966, 21:345-350.
  • Mortimer EA Jr, “DTP and SIDS: when data differ”, Am J Public Health 1987 Aug; 77(8):925-926.

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Vaccines and Metabolism

  • Deutsch J, ” [Temperature changes after triple-immunization in infant age],” Padiatr Grenzgeb 1976;15(1):3-6. [Article in German]
  • NA, “[Temperature changes after triple immunization in childhood],” Padiatr Grenzgeb 1976;15(1):7-10. [Article in German]
  • [Considering that the thyroid controls our Basal Metabolism, it would appear that vaccines altered (depressed) thyroid activity.] Vaccines Altering Resistance to Disease:
  • Burmistrova AL, “[Change in the non-specific resistance of the body to influenza and acute respiratory diseases following immunization diphtheria-tetanus vaccine],” Zh Mikrobiol Epidemiol Immunobiol 1976; (3):89-91. [Article in Russian] Vaccinations and Deafness Citations: So I did a background check to see if there was any scientific evidence linking vaccines to deafness and hearing loss. Here are some of the articles I found:
  • Kaga, “Unilateral Total Loss of Auditory and Vestibular Function as a Complication of Mumps Vaccination”, Int J Ped Oto, Feb 1998, 43(1):73-73
  • Nabe-Nielsen, Walter, “Unilateral Total Deafness as a Complication of the Measles- Mumps- Rubella Vaccination”, Scan Audio Suppl, 1988, 30:69-70
  • Hulbert, et al, “Bilateral Hearing Loss after Measles and Rubella Vaccination in an Adult”, NEJM, 1991 July, 11;325(2):134
  • Healy, “Mumps Vaccine and Nerve Deafness”, Am J Disorder Child, 1972 Jun; 123(6):612
  • Jayarajan, Sedler, “Hearing Loss Following Measles Vaccination”, J Infect, 1995 Mar; 30(2):184-185
  • Pialoux, P et al, “Vaccinations and Deafness”, Ann Otolaryng (Paris), Dec 1963, 80:1012-1013.
  • Angerstein, W, et al, “Solitary Hearing and Equilibrium Damage After Vaccinations”, Gesundheitswesen, May 1995, 57(5): 264-268.
  • Brodsky, Stanievich, “Sensorineural Hearing Loss Following Live Measles Virus Vaccination”, Int J Ped Oto, 1985 Nov; 10(2):159-163
  • Koga, et al, “Bilateral Acute Profound Deafness After MMR Vaccination- Report of a Case”, Nippon Jibiin Gakkai Kai, 1991 Aug;94(8):1142-5
  • Seiferth, LB, “Deafness after Oral Poliomyelitis Vaccination – a Case Report and Review”, HNO, 1977 Aug; 25(8): 297-300
  • Pantazopoulos, PE, “Perceptive Deafness Following Prophylactic use of Tetanus anittoxin”, Laryngoscope, Dec 1965, 75:1832-1836.
  • Zimmerman, W, “Observation of a case of Acute Bilateral Hearing Impairment Following Preventive Poliomyelitis Vaccination (type 3)”, Arch Ohr Nas Kehlkopfheilk, 1965, 185:723-725. Vaccinations and Kidney Disorders Citations:
  • Jacquot, C et al, “Renal Risk in Vaccination”, Nouv Presse Med, Nov 6, 1982, 11(44):3237-3238.
  • Giudicelli, et al, “Renal Risk in Vaccination”, Presse Med, Jun 11, 1982, 12(25):1587-1590.
  • Tan, SY, et al, “Vaccine Related Glomerulonephritis”, BMJ, Jan 23, 1993, 306(6872):248.
  • Pillai, JJ, et al, “Renal Involvement in Association with Post-vaccination Varicella”, Clin Infect Disorder, Dec 1993, 17(6): 1079-1080.
  • Eisinger, AJ et al, “Acute Renal Failure after TAB and Cholera Vaccination”, B Med J, Feb 10, 1979, 1(6160):381-382.
  • Silina, ZM, et al, “Causes of Postvaccinal Complications in the Kidneys in Young Infants”, Pediatria, Dec 1978, (12):59-61.
  • Na, “Albuminurias”, Concours Med, Mar 1964, 85:5095-5098. [vaccination adverse reactions]
  • Oyrl, A, et al, “Can Vaccinations Harm the Kidney?”, Clin Nephrol, 1975, 3(5):204-205.
  • Mel’man Nia, “[Renal lesions after use of vaccines and sera].” Vrach Delo 1978 Oct;(10):67-9, [Article in Russian]
  • Silina ZM, Galaktionova TIa, Shabunina NR, “[Causes of postvaccinal complications in the kidneys in young infants].” Pediatriia 1978 Dec;(12):59-61, [Article in Russian]
  • Silina EM, et al, “[Some diseases of the kidneys in children during the 1st year of life, following primary smallpox vaccination and administration of pertusis-diphtheria-tetanus vaccine].” Vopr Okhr Materin Det 1968 Mar; 13(3):79-80, [Article in Russian]

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Vaccines and Skin Disorders 

  • Illingsworth R, Skin rashes after triple vaccine,” Arch Dis Child 1987 Sep; 62(9):979.
  • Lupton GP, “Discoid lupus erythematosus occurring in a smallpox vaccination scar,” J Am Acad Dermatol, 1987 Oct; 17(4):688-690.
  • Kompier, A J, “Some Skin Diseases caused by Vaccinia Virus [Smallpox],” Ned Milt Geneesk T, 15:149-157, May 1962.
  • Weber, G et al, “Skin Lesions Following Vaccinations,” Deutsch Med Wschr, 88:1878-1886, S7 Sept 1963.
  • Copeman, P W, “Skin Complications of Smallpox Vaccination,” Practitioner, 197:793-800, Dec 1966.
  • Denning, DW, et al, “Skin Rashes After Triple Vaccine,” Arch Disorder Child, May 1987, 62(5): 510-511. Vaccinations and Abcesses:
  • Sterler, HC, et al, “Outbreaks of Group A Steptococcal Abcesses Following DTP Vaccination”, Pediatrics, Feb 1985, 75(2):299-303.
  • DiPiramo, D, et al, “Abcess Formation at the Site of Inoculation of Calmette-Guerin Bacillus (BCG),” Riv Med Aeronaut Spaz, Jul-Dec 1981, 46(3-4):190-199. Vaccinations and Shock:
  • Caileba, A et al, “Shock associated with Disseminated Intravascular Coagulation Syndrome following Injection of DT.TAB Vaccine, Prese Med, Sept 15, 1984, 13(3):1900. Vaccines: The Weird, The Wild and The Hilarious Citations: Sometimes there are articles published about the strangest facts related to vaccines that defies our imagination and ability to understand them. They were written seriously by well-meaning scientific persons, but their titles can be seen differently. Some are funny, some are sad and some are purely scientific folly. See if you can figure these out:
  • Pathel, JC, et al, “Tetanus Following Vaccination Against Small-pox”, J Pediatr, Jul 1960; 27:251-263. [Now you need a tetanus vaccination!]
  • Favez, G, “Tuberculous Superinfection Following a Smallpox Re-Vaccination”, Praxis, July 21, 1960; 49:698-699. [Super means large/big/great!]
  • Bonifacio, A et al, “Traffic Accidents as an expression of “Iatrogenic damage”, Minerva Med, Feb 24, 1971, 62:735-740. [But officer I was just vaccinated!]
  • Baker, J et al, “Accidental Vaccinia: Primary Inoculation of a Scrotum”, Clin Pediatr (Phila), Apr 1972, 11:244-245. [Ooops, the needle slipped.]
  • Edwards, K, “Danger of Sunburn Following Vaccination”, Papua New Guinea Med J, Dec 1977, 20(4):203. [Are vaccines phototoxic?]
  • Stroder, J, “Incorrect Therapy in Children”, Folia Clin Int (Barc), Feb 1966, 16:82-90. [Agreed.]
  • Wehrle PF, “Injury associated with the use of vaccines,” Clin Ther 1985;7(3):282-284. [Dah!]
  • Alberts ME, “When and where will it stop”, Iowa Med 1986 Sep; 76(9):424. [When!]
  • Breiman RF, Zanca JA, “Of floors and ceilings — defining, assuring, and communicating vaccine safety”, Am J Public Health 1997 Dec;87(12):1919-1920. [What is in between floors and ceilings?]
  • Stewart, AM, et al, “Aetiology of Childhood Leukaemia”, Lancet, 16 Oct, 1965, 2:789-790.
  • Nelson, ST, “John Hutchinson On Vaccination Syphilis (Hutchinson, J)”, Arch Derm, (Chic), May 1969, 99:529-535. [Vaccinations and STDs!]
  • Mather, C, “Cotton Mather Anguishes Over the Consequences of His Son’s Inoculation Against Smallpox”, Pediatrics, May 1974; 53:756. [Is it for or against?]
  • Thoman M, “The Toxic Shot Syndrome”, Vet Hum Toxicol, Apr 1986, 28(2):163-166. [Animals are not exempt from vaccination damage either!]
  • Johnson, RH, et al, “Nosocomial Vaccinia Infection”, West J Med, Oct 1976, 125(4):266-270. [Nosocomial means a disease acquired in a doctor’s office or hospital.]
  • Heed, JR, “Human Immunization With Rabies Vaccine in Suckling Mice Brain,” Salud Publica, May-Jun 1974, 16(3): 469-480. [Have you had your suckling mice brains today?]
  • Tesovic, G et al, “Aseptic Meningitis after Measles, Mumps and Rubella Vaccine”, Lancet, Jun 12, 1993, 341(8859):1541. [AM has same symptoms as poliomyelitis!]
  • Buddle, BM et al, “Contagious Ecthyma Virus-Vaccination Failures”, Am J Vet Research, Feb 1984, 45(2):263-266.
  • Freter, R et al, “Oral Immunization And Production of Coproantibody in Human Volunteers”, J Immunol, Dec 1963, 91:724-729. [Guess what copro- means …. Feces.]
  • NA, “Vaccination, For and Against”, 1964, Belg T Geneesk, 20:125-130. [Is it for or against?]
  • Sahadevan, MG et al, “Post-vaccinal Myelitis”, J Indian Med Ass, Feb 16, 1966, 46:205-206. [Did I mention myelitis?]
  • Castan, P et al, “Coma Revealing an acute Leukosis in a child, 15 days after an Oral Anti-poliomyelitis Vaccination,” Acta Neurol Bekg, May 1965, 65:349-367. [Coma from vaccines!]
  • Stickl, H, et al, “Purulent [pus] meningitides Following Smallpox Vaccination. On the Problem of Post- Vaccinal Decrease of Resistance”, Deutsch Med Wschr, Jul 22, 1966, 91:1307-1310. [Vaccines are the injection of viruses cultured from pus …]

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The Polio Vaccine And Cancer 

  •  Shah, K and Nathanson, N. “Human exposure to SV40.” American Journal of Epidemiology, 1976; 103: 1-12.
  • Innis, M.D. “Oncogenesis and poliomyelitis vaccine.” Nature, 1968; 219:972-73.
  • Soriano, F., et al. “Simian virus 40 in a human cancer.” Nature, 1974; 249:421-24.
  • Weiss, A.F., et a;. “Simian virus 40-related antigens in three human meningiomas with defined chromosome loss.” Proceedings of the National Academy of Science 1975; 72(2):609-13.
  • Scherneck, S., et al. “Isolation of a SV-40-like papovavirus from a human glioblastoma.” International Journal of Cancer 1979; 24:523-31.
  • Stoian, M., et al. “Possible relation between viruses and oromaxillofacial tumors. II. Research on the presence of SV40 antigen and specific antibodies in patients with oromaxillofacial tumors.” Virologie, 1987; 38:35-40.
  • Stoian, M., et al. “Possible relation between viruses and oromaxillofacial tumors. II. Detection of SV40 antigen and of anti-SV40 antibodies in patients with parotid gland tumors.” Virologie, 1987; 38:41-46.
  • Bravo, M.P., et al. “Association between the occurrence of antibodies to simian vacuolating virus 40 and bladder cancer in male smokers.” Neoplasma, 1988; 35:285-88.
  • O’Connell, K., et al. “Endothelial cells transformed by SV40 T-antigen cause Kaposi’s sarcoma-like tumors in nude mice.” American Journal of Pathology,1991; 139(4):743-49.
  • Weiner, L.P., et al. “Isolation of virus related to SV40 from patients with progressive multifocal leukoencephalopathy.” New England Journal of Medicine, 1972; 286:385-90.
  • Tabuchi, K. “Screening of human brain tumors for SV-40-related T-antigen.” International Journal of Cancer 1978; 21:12-17.
  • Meinke, W., et al. “Simian virus 40-related DNA sequences in a human brain tumor.” Neurology 1979; 29:1590-94.
  • Krieg, P., et al. “Episomal simian virus 40 genomes in human brain tumors.” Proceedings of the National Academy of Science 1981; 78:6446-50.
  • Krieg, P., et al. “Episomal Simian Virus 40 Genomes in Human Brain Tumors.” Proceedings of the National Academy of Sciences of the USA, 1981, 78(10):6446-6450.
  • Krieg, P., et al. “Cloning of SV40 genomes from human brain tumors.” Virology 1984; 138:336-40.
  • Geissler, E. “SV40 in human intracranial tumors: passenger virus or oncogenic ‘hit-and-run’ agent?” Z Klin Med, 1986; 41:493-95.
  • Geissler, E. “SV40 and Human Brain Tumors.” Progress in Medical Virology, 1990; 37:211-222.
  • Bergsagel, D.J., et al. “DNA sequences similar to those of simian virus 40 in ependymomas and choroid plexus tumors of childhood.” New England Journal of Medicine, 1992; 326:988-93.
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Reviewed/Updated: 09/14
Content Created: 03/14  

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